Accidents of eruption and evolution of teeth

Accidents of eruption and evolution of teeth

Accidents of eruption and evolution of teeth.

I- Introduction: 

Dental eruption is a physiological phenomenon that allows a tooth to erupt into the oral cavity to a functional position. It involves in particular bone resorption phenomena. 

The eruption of permanent teeth is preceded by the resorption of temporary teeth up to the premolars. 

Eruption abnormalities occur in both temporary and permanent teeth, although their nature is usually different. 

Dental development accidents are pathological manifestations of a local, regional or general nature which precede, accompany or follow the eruption of temporary or permanent teeth. 

II – Definitions: 

Dentition: or dental evolution is an active biological phenomenon that is part of the general growth of the individual. It is the placement of teeth on the arch. It is a dynamic phenomenon. 

Denture: this is the set of dental organs present on the arch. It is a static phenomenon. 

III- The mechanisms of dental eruption:

The actual eruption of the tooth begins in principle when the first quarter of the root is built (Van Der Linden, 1983). This is a complex mechanism, comprising several stages, not yet completely elucidated to date. This mechanism ensures both the formation of the root and its periodontal environment, the intraosseous displacement of the germ, the emergence of the crown in the oral cavity as well as its path to the occlusal plane.

Pre-eruptive phase  : 

It corresponds to the period of pre-eruptive movements within the alveolar bone during the pre-eruptive movements, the bone crypts are transformed and move with the germs. The ceiling of each of them – or wall of the cavity oriented towards the occlusion plane – is pierced by an orifice which corresponds to the entrance of the gubernacular canal. 

This bony tunnel connects the crypt to the cortex, and sometimes to the alveolar wall of the deciduous tooth. This is the path that the tooth takes during its eruption. 

Pre-functional eruptive phase

Once the crown is formed, eruption begins. This movement coincides with the start of root formation. The developing tooth begins to move through the alveolar bone through the polarized phenomenon of bone apposition/resorption. 

The mucosa is usually breached when root growth reaches half or two-thirds of the definitive root length. The mucosa is usually breached when root growth reaches half or two-thirds of the definitive root length.

Post-eruptive functional phase: 

 After emerging into the oral cavity, the tooth continues its eruption until it comes into contact with the opposing teeth and gains a functional position. 

The eruption will continue throughout life. This is the post-eruptive phase. 

VI – Temporary dentition:

a/ Chronology of the eruption of temporary teeth:

The eruption of temporary teeth in a healthy infant occurs in groups of teeth per semester: 

  • Incisors: from the 6th to   the 12th   month
  • 1st   Molars: from the 12th to the 18th   month
  • Canines: from the 18th to the 24th   month
  • 2nd   Molars: from the 24th to the 30th   month

* Order of eruption of central and lateral incisors > and < .

  • Lower Central Inc.
  • Superior Central Inc.
  • Upper side inc.
  • Lower side inc.

b/ Eruption accidents: 

1/ Early eruptions:

Common in the lower incisors

*We blame: – a hereditary family trait

                              – infection during pregnancy

                              – an endocrine factor

*We distinguish: vestigial teeth which only persist on the arch a few days after birth.

2/ Delayed eruptions: 

  • When it is an isolated tooth: the cause is local: trauma or malformation. 
  • When several teeth are affected: the cause is general: endocrinopathy, vitamin deficiency, heredity.

3/ Local accidents

Several theories have been put forward to explain these phenomena:

  • Infectious theory: infection or irritation of the pericoronary sac;
  • Reflex theory: local irritation at the level of the trigeminal nerve would cause remote manifestations by reflex;
  • Organic predisposition theory: Some people are more likely to have accidents than others.   
  • Other causes: heredity, endocrine diseases, hygiene, maternal diseases, diseases of early childhood could intervene. 

a- Gingivo-dental pruritus: 

  • The infant bites his lips and 
  • Drooling due to hypersalivation (sialorrhea). 
  • The gum is inflamed at the site of future eruption. 

b- Pericoronitis:

It is the inflammation and infection of the pericoronary sac:

– Abundant salivation;

– Red, swollen, painful, sometimes purulent gums;

– Hyperthermia and 

– Hustle.

c- Stomatitis:

       The inflammation can spread to the neighboring mucosa and determine all the aspects of stomatitis:

 * Aphthoid stomatitis 

 * Stomatitis of primary herpes infection. 

d- Pericoronal eruption cyst or follicular cyst
– Usually located at the level of the 2nd temporary   molar or on an edentulous gingival ridge.
– It appears as a rounded, bluish, translucent, fluctuant and painless swelling.
– Its content is a stringy, citrine liquid.
– The X-ray shows a radiolucent image around the crown of the tooth.
* treatment: cystotomy (marsupialization). The neighboring germ must be respected.

e – Expulsive folliculitis:

– Following an inflammatory phenomenon or a local infection, the dental follicle is eliminated spontaneously. 

– A fleshy bud then remains which will disappear as well as the local signs.

– Can sometimes be complicated by osteomyelitis 

– Treatment: Antibiotic therapy + local care.

– Langerhans cell histiocytosis must be ruled out in the presence of any expulsive folliculitis. 

4/ Regional accidents:

– erythrosis jugal (tooth burn)

– hypersalivation, 

– tearing, 

– serous nasal discharge

5/ General accidents:

– Benign and punctuated by eruptions

We can distinguish:

      – thermal disorders: hyperthermia for 48 hours.

      – nervous disorders: agitation, insomnia, convulsions. 

      – digestive disorders: vomiting, loss of appetite, diarrhea, slowed growth and height and weight disorders 

      – respiratory problems: dry or whooping cough accompanying the rash; sometimes bronchitis and bronchoalveolitis. 

6/ Retention of temporary teeth:

– May affect one or more teeth (especially molars)

– Radiologically we can observe:

 * rhizal analysis of the retained tooth

 * an absence of the corresponding permanent tooth

* a retained tooth stuck between two closely spaced permanent teeth

– The evolution is towards spontaneous fall, inclusion, infectious accident. 

– Maintaining this retained tooth will cause permanent tooth misalignments on the arch.

– Treatment: extraction of the affected tooth followed by ODF treatment

It is mandatory to ensure the existence of the permanent tooth (by X-ray) before extracting the retained analogous temporary tooth.

In case of generalized delay it could be due to a vitamin deficiency, an endocrinopathy or a hereditary disease. 

 – Therapeutic: 

1- Local symptomatic treatment in common accidents of temporary tooth eruption based on analgesics (paracetamol), anti-inflammatories (Niflumic acid: nifluril®, ibuprofen).

2- Good hygiene, disinfection, massage and friction using anesthetic balms.

3- Avoid incisions except in cases of pericoronitis;

4- Power monitoring. 

V/ Permanent dentition: 

A- Chronology of the eruption of permanent teeth:

After the temporary crown falls out, the crown of the permanent tooth replaces it and migrates axially until it meets its antagonist.

The eruption of permanent teeth is annual: one group of teeth per year.

  • 6 years: 1st   molar
  • 7 years: central incisor
  • 8 years: lateral incisor
  •  9 years: 1st   premolar
  • 10 years: canine
  • 11 years old: 2nd   premolar
  • 12 years: 2nd   molar

B- Accidents of permanent dentition 

1/ Early teething:

She is exceptional. 

It must be preceded by the loss of temporary teeth.

2/ Delayed teething can be caused by:       

  a) local causes:

  – Persistence of temporary teeth due to irregular or late rhizalysis

  – Closure of diastemas after early extraction of temporary teeth.

  – Radiculo-dental cyst pushing back the germ of the permanent tooth.

  – Pericoronal cyst of the permanent tooth.

  – Presence of a local tumor hindering the migration of the temporary or permanent tooth (odontoma).

3/ Difficult teething:

These are complicated eruptions of evolutionary accidents. All teeth are affected, but especially the lower wisdom tooth. It can occur on teeth in a normal or abnormal position. 

Teething becomes difficult when:

a) Infectious accidents:

In children, these infectious accidents mainly accompany the included canines:

– rhinitis, sinusitis, osteitis

– neuralgia radiating to the sinus and eye

– a neighborhood infection 

1) Pathogenesis:

These accidents are explained by:

– morphological anomaly of the root, corona, obliquity of the tooth.

– superinfection of physiological pericoronitis of eruption.

– microbial penetration into the pericoronal sac when it opens into the oral cavity.

2) Symptoms:

Accidents occur between 18 and 25 years of age during the DDS eruption period.

1/ The initial accident: Acute pericoronitis:

It is the infection of the pericoronary sac and the adjoining mucosa.

The diagnosis is based on:                            

* Pain: spontaneous, or caused by chewing by bruising of the mucous membrane, on palpation of the mucous hood. 

* Dysphagia 

* Trismus: more or less severe, tight.

* The clinical examination found: 

* a congested, edematous mucosa, marked by the imprint of the opposing tooth.

* a drop of pus on local pressure.

* part of the crown is visible sometimes surrounded by ulcerated mucosa.

* X-ray: specifies the morphology of the tooth, its relationship with the lower dental nerve, with the neighboring tooth, its inclination, its possible enclavement, the prior existence of a possible cyst.

* The evolution:

– is done either towards temporary remission (medical TTT);

– or towards complicated forms (cellular accidents or general accidents, septicemic).

* Treatment: 

Extraction in case of repeated accidents, cysts, cavities or misaligned teeth.

2/ Mucosal accidents:
this is unilateral odontiatic stomatitis related to the development of the wisdom tooth which can be erythematous or ulcerative.

3/Lymph node accidents can be complicated by periadenitis and adenophlegmon.

4/ Cellular accidents: extension of the infection from the pericoronary sac.

5/ Bone accidents: exceptional osteitis.

6/ Remote infection: the spread of pericoronary infection by lymphatic or vascular route will graft itself to the kidneys (glomerulonephritis); the heart (bacterial endocarditis); the eye (uveitis), the joints, etc. 

b) Mechanical accidents:

– Fractures, dento-maxillary disharmonies, articulation disorders with repercussions on the TMJ 

c) Nervous and reflex accidents:

  • Sensitive: skin hypoesthesia, facial pain, paresthesia, ringing in the ears, etc.
  • Synalgia: pain is felt in other mandibular or maxillary teeth. 

– Motor: paralysis, facial spasms, trismus 

– Sensory: eye or hearing problems. 

d) Tumor disorders:

– Pericoronary (follicular) cyst

– Eruptive cyst (Epstein pearl).

4/ The treatment: 

It depends on the tooth environment as follows:

* The tooth will be saved if:

  •  The space to accommodate it is sufficient
  •  There are no obstacles to its development.
  •  Its position allows it to erupt correctly 

* The tooth will be extracted if:

  • The X-ray shows that it cannot erupt completely (insufficient space, impacted tooth, cyst, tumor, etc.)
  • The tooth is the cause of infectious or mechanical accidents. It is recommended to do this extraction at a distance from the infectious episode (antibiotic therapy). 

VI – Dental inclusions:

A) – Definition:

There is dental inclusion when:

– the tooth does not erupt on the expected date

– the pericoronary sac is not in communication with the oral cavity 

– the tooth is located near its usual seat.

Inclusions can affect one tooth or a group of teeth (multiple inclusions).

B) – Differential diagnosis:

  •  Disimpacted tooth: the pericoronal sac is open in the oral cavity . The apices are closed. 
  • Retained tooth: there is localized delay in eruption
  •  Impacted tooth: an obstacle hinders the normal eruption of the tooth.

C) – Frequency:

Inclusion in descending order: DDS >,  canine > , premolars, incisor < , 1st molar < .

D) – Etiopathogenesis:

We are mainly talking about:

– An abnormality of the pericoronary sac;

– A coronary or radicular malformation;

– An obstacle: cyst, persistence of the temporary tooth;

– A tumor.

E) – Included canine:

The cause of this inclusion is often related to an obstacle, a malformation, a malposition of the germ.

Accidents of eruption and evolution of teeth

She is suspected of:

  • a palatal or vestibular arch.
  • an inter-incisal diastema.
  • a displacement of the lateral incisor.

It is most often revealed by infectious and/or mechanical accidents 

Canine inclusion is confirmed by:

A radiological check (panoramic, occlusal and retroalveolar ) . 

F) – Upper central incisor:

Its inclusion is due to trauma to the germ. 

I) – Therapeutic attitude:

  • Generally, an impacted, clinically silent tooth does not need to be extracted.
  • However, in the context of ODF treatment, or prosthetic restoration, there may be an exception, if not an obligation.
  • Extraction is the rule when the tooth becomes symptomatic or when accidents recur. 

Accidents of eruption and evolution of teeth

VI – Conclusion:

Tooth eruption often occurs gradually and without any particular problem. However, it is possible that in some cases, temporary or permanent teeth are accompanied by local, locoregional or general signs that the dental practitioner must master for the most appropriate therapeutic management of the patient. 

Accidents of eruption and evolution of teeth

 Bibliographic references:

  1. Vaysse F, Noirrit E, Bailleul-Forestier I, Bah A, Bandon D. Anomalies of dental eruption.  Archives of pediatrics 2010; 17: 255-257. 
  2. Moulis E, Favre de Thierens C, Goldsmith MC, Torres JH. Anomalies of the eruption. EMC (Elsevier Masson SAS, Paris), Stomatology/Odontology, 22-032-A10. 2002.
  3. Peron M. Accidents of evolution of wisdom teeth. EMC (Elsevier Masson SAS, Paris), Stomatology/Odontology, 22-032-E10. 2003.
  4. Haroun, A., HAS recommendations on the avulsion of third molars. Bulletin of the National Union for the Interest of Dentofacial Orthopedics, 2008(37); p. 22-37.
  5. Talibart, F., Relevance of indications for avulsion of third molars in Aquitaine, in College of Health Sciences 2016, University of Bordeaux UFR of Odontological Sciences.
  1. Piette E, Goldberg M. The normal and pathological tooth. Brussels: De Boeck University; 2001.
  2. Accidents of eruption and evolution of teeth

  Baby teeth need to be taken care of to prevent future problems.
Periodontal disease can cause teeth to loosen.
Removable dentures restore chewing function.
In-office fluoride strengthens tooth enamel.
Yellowed teeth can be treated with professional whitening.
Dental abscesses often require antibiotic treatment.
An electric toothbrush cleans more effectively than a manual toothbrush.
 

Accidents of eruption and evolution of teeth

Accidents of eruption and evolution of teeth

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